Categories
Blog

Why I’m Training My Clients (and Myself) to Walk Well for Brain Health and Longevity

Why I’m Training My Clients (and Myself) to Walk Well for Brain Health and Longevity

Have you ever thought about how fast you walk? Probably not, unless you are on a hike with someone who walks faster than you.  Then all you can think about is keeping up and how do their legs make them move faster than you even though they appear shortere!

Over 4 decades of competing sports, makes me a natural tracker of performance metrics. But now I’m not evaluating myself against people in their 20’s…yes I still do that…but people in the 80’s!

As a clinician working in lifestyle medicine and human performance, I constantly look at how biomechanics intersect with systemic health. It turns out that how fast and efficiently you walk is one of the most accurate windows we have into your biological age, neurological health, and lifespan.

The Science Behind the ‘Super-Mover’

A study published in Neurology reveals that older adults who maintain a brisk, robust gait—dubbed “super-movers”—don’t just have healthier joints and muscles; they exhibit remarkable cognitive resilience.

Dr. Joe Verghese and his team analyzed data from major aging cohorts involving thousands of older adults (average age 84). The results were clear:

  • Dementia Risk: Super-movers were roughly 50% less likely to experience cognitive decline and 60% less likely to be diagnosed with Alzheimer’s disease or related dementias compared to their slower-walking peers over a multi-year follow-up.
  • Brain Structure: Neuroimaging showed super-movers preserved significantly greater volume in the hippocampus—the brain’s central hub for memory and spatial navigation.
  • Cognitive Reserve: Remarkably, post-mortem analyses revealed that even when super-movers had structural brain changes associated with Alzheimer’s, they frequently remained symptom-free in life. Their physical capacity helped build a buffer—a cognitive reserve.

Why Gait Speed Is So Useful

In clinical practice, I’ve heard people suggest that walking speed carries as much predictive weight as blood pressure, heart rate, or blood glucose…having said that when I brought it up with my doctor he brushed it off with a shrug…so it hasn’t reached grass roots medicine yet. 

Walking isn’t just a basic mechanical task; it is a complex neurological event. To walk rapidly and with a balanced stride, your central nervous system, cardiovascular system, and musculoskeletal structures must operate in complete synchronization.

  • Early Warning Indicator: Changes in gait dynamics—such as a shortened stride, decreased velocity, or subtle asymmetry—can show up to seven years before clinical symptoms of dementia appear (Verghese et al.).
  • Cancer and Cardiovascular Protection: Large-scale population data from the UK Biobank (tracking over 335,000 adults) demonstrated that individuals walking at a brisk pace (~5–6 km/h) experienced significantly lower risks for multiple types of cancer. Furthermore, University of Sydney research showed that a brisk walking pace reduced all-cause mortality by 24%, with cardiovascular mortality risk dropping by over 50% in older demographics.

How Brisk Walking Protects the Nervous System

When you increase your walking speed, several metabolic and neurological cascades occur simultaneously:

  1. Neurotrophic Factor Release: Fast walking stimulates the production of Brain-Derived Neurotrophic Factor (BDNF), a key protein that promotes neuroplasticity, protects existing neurons, and encourages new synaptic connections.
  2. Myokine Signaling: Your lower body contains the largest muscle groups in human anatomy. When these muscles contract dynamically during brisk walking, they secrete specialized signaling peptides called myokines into the bloodstream, which reduce systemic inflammation and support brain cell survival.
  3. Cerebral Blood Flow: Higher-intensity movement increases pulsatile blood flow to the brain, optimizing oxygen delivery and nutrient clearance via the glymphatic system.

Test Your Gait: The 5-Meter Speed Check

In clinical settings, we often use short-distance velocity tests to calculate functional baseline speed. You can easily test this yourself:

  1. Measure out 5 meters on a flat, straight surface.
  2. Time how many seconds it takes you to walk that distance at your standard, purposeful pace.
  3. Divide 5 meters by your time in seconds to get your speed in meters per second (m/s).
  • The Benchmark: A speed above 1.0 m/s (taking under 5 seconds to cover 5 meters) correlates with healthy biological aging and strong functional capacity.
  • The Threshold: A gait speed dropping below 0.8 m/s indicates increased risk for physical frailty, fall hazards, and accelerated cognitive decline.

Note: Even incremental improvements matter—research shows that increasing your average gait speed by as little as 0.1 m/s correlates with measurable gains in functional independence and healthspan.

How To Improve Gait & Velocity

If you want to increase your walking speed and optimize your movement mechanics, focus on these structural and habits-based interventions:

  • Restore Foot Mechanics & Toe Splay: Years of restrictive footwear narrow the forefoot, limiting the ground contact area of the hallux (big toe) and reducing propulsive power. Utilizing toe spacers and spending time barefoot on natural surfaces improves toe alignment, arch activation, and ground force transmission. Personally I like the company correct toes and used to exclusively sell their product in my clinic. 
  • Build Lower Leg Power (Single-Leg Calf Raises): An efficient stride requires strong ankles. Stand on one leg and perform slow, controlled calf raises through full range of motion. Aim to comfortably complete 20 continuous repetitions per side without significant compensation.
  • Test Big Toe Engagement: Sit in a chair, place a small card under your big toe, and press down firmly into the floor without clawing your toes. Have someone pull the card; if it slides out easily, focus on strengthening your intrinsic foot flexors and posterior tibialis muscle.
  • Integrate Retro (Backward) Walking: Walking backward forces a different neuromuscular coordination pattern, increases quadriceps and tibialis anterior recruitment, reduces knee joint stress, and sharpens proprioception. Start on a flat, clear path or a powered-off treadmill for 3–5 minutes.
  • Maintain Postural Alignment: An upright posture optimizes biomechanical efficiency, opens the thoracic cavity for full diaphragmatic respiration, and reduces unnecessary energy expenditure through the kinetic chain.

References

  1. Verghese, J., et al. (2026). Gait performance, super-movers, and risk of cognitive decline in older adults. Neurology.
  2. Abellana, R., et al. (2017). Gait speed as a predictive biomarker for cognitive impairment and dementia onset. Journal of Alzheimer’s Disease, 56(4), 1421-1429.
  3. Stamatakis, E., et al. (2018). Self-rated walking pace and all-cause, cardiovascular disease and cancer mortality: individual participant pooled analysis of 50,225 walkers. British Journal of Sports Medicine, 52(12), 761-768.
  4. Zhai, X., et al. (2022). Association of walking pace with incident cancer risk: A prospective cohort study of 337,000 UK Biobank participants. European Journal of Epidemiology.
  5. Pedersen, B. K. (2019). Physical activity and muscle-brain crosstalk: myokines and BDNF in cognitive preservation. Nature Reviews Endocrinology, 15(7), 383-392.
Categories
Podcasts

Podcast Episode 64 – Understanding Menopause

Podcast Episode 64 – Katie Lomas

🎙️ Understanding Menopause with Kate Lomas | Episode 64

Menopause is one of the most significant transitions in a woman’s life, yet it’s often misunderstood. In this episode, Ed Paget sits down with menopause specialist Kate Lomas to break down the myths, explain what’s really happening during perimenopause and menopause, and discuss how hormones impact everything from bone health and heart health to mood, weight, energy, and longevity.

You’ll discover:
– The difference between perimenopause and menopause
– Why symptoms can be worse before menopause
– The truth about hormone replacement therapy (HRT)
– Bone health, osteoporosis, and aging well
– Weight gain, metabolism, and hormone changes
– Common myths about breast cancer and HRT
– Practical strategies to support long-term health and vitality

Whether you’re navigating this stage yourself or want to better understand what the women in your life are experiencing, this conversation is packed with valuable insights and practical advice.

🎧 Listen to the full episode on your favorite podcast platform.
📺 Watch the full interview on YouTube for the complete conversation.
💬 Have a question or experience with perimenopause or menopause? Share it in the comments—we’d love to hear from you!

Watch the full podcast episode video here  or audio episode.

Categories
Blog

Scoliosis Genes and Nutrition

How Your Genes and Nutrition Play A Roll In Your Scoliosis

Traditional scoliosis care often traps families in a cycle of “wait and see,” watching a curve get worse while feeling powerless to stop it. For years, the industry treated Adolescent Idiopathic Scoliosis (AIS) as a simple mechanical failure—like a leaning tower that just needs a bigger brace to push it back.

The reality is that if we only look at the bones, we could be missing the “software” glitch in the brain and DNA that is driving the “hardware” collapse. New research suggests shifting from passive observation to a neuro-genomic intervention, this means we would stop treating the symptom and start addressing the body’s internal signaling.

1. The Genetic “Software” Glitch

Many teens face rapid curve progression during puberty that seems “unstoppable” even with a brace. This happens because their DNA is sending the wrong instructions to their ligaments and joints.

Researchers have begun to look at the genes for the solution. WHat they discovered is that people with scoliosis show some different gene variants when compared to people without.  Two common variants are the COMT and MTHFR gene variants. A COMT variant slows down how your body clears out estrogen, which leads to “stretchy” joints (ligamentous laxity). When you combine this with an MTHFR “bottleneck” that starves the brain of nutrients, the spine loses its internal support system. Research shows that when these two variants collide, there is a greater than 20° progression in over 80% of cases.

By identifying these markers early through genomic testing, we can use targeted support like L-Methylfolate and SAMe to clear the metabolic path. The aim here is to stabilize the body’s chemistry before the growth spurt causes the curve to win. But are there other genes that could predict scoliosis progression?

 

2. The Brain’s Blind Spot

If your brain has low postural memory, it essentially “forgets” where the spine is in space. This makes it hard to fix a posture that the brain doesn’t realize is crooked. I’ve heard reports of when a tether breaks in someone’s back their curve often regresses to what it was. Meaning the brain’s internal map of the spine isn’t straight. 

This is often driven by a “triple-threat” of chemical imbalances: low serotonin, low histamine, and high norepinephrine. For instance, a “hyper-utilizer” MAOA variant acts like a vacuum, sucking up serotonin—the “posture manager”—before it can tell the muscles to hold the spine upright.

When we balance these chemicals (using tools like 5-HTP), we restore the brain’s “map” of the body. This may just give the nervous system the clarity it needs to maintain a straight, stable spine automatically. But how do genes play a role in things like bone building?

 

3. The Brittle Foundation

A spine cannot stay straight if the “bricks” used to build it are weak. Many AIS patients suffer from low bone density, making their spine “spongy” and susceptible to bending under pressure.

This isn’t just about calcium; it’s about the management of that calcium. Vitamin D acts as the delivery truck that brings calcium into the blood, but without Vitamin K2 acting as the driver, that calcium never reaches the bone matrix. It just floats around, leaving the spine weak during critical growth periods.  Some studies show that up to 90% of teens with scoliosis are low in Vitamin D

Optimizing Vit D and K2  ensures that as the teen grows, they are building a high-density, resilient skeletal frame that can actually resist the mechanical pull of a curve.

 

4. The Integrated Path Forward

Relying solely on external exercises (outside-in) often feels like fighting a losing battle against your own biology (inside-out).

Traditional Scoliosis Specific exercises are essential, but they are often performed on a body that is chemically “unbalanced.” It’s like trying to align the wheels on a car while the frame is still bent

This is where the proponents of Genomic-Informed Rehab come in. By combining neuromuscular re-education (like the Scoliosis Correction Protocol with targeted nutrition to fix the internal chemistry, you stop being a victim of your genetics. You become the architect of your own spinal health, with a treatment plan that is as unique as your DNA.

 

References

  • Blasco-Fontecilla, H. (2023). Journal of Clinical Medicine.
  • Janusz, P., et al. (2014). Medical Science Monitor.
  • Morningstar, M. (2011). Scoliosis and Spinal Disorders.
  • Morningstar, M. (2023). Serotonin – Neurotransmitter and Hormone.
  • Morningstar, M. (2024). Medical Research Archives.
  • Nicotera, A. G., et al. (2021). Brain Sciences.
  • Wright, J., et al. (2025). Journal of Genomics.